Abstract: A system and method for treating a necrotic section of bone. At least one channel is drilled into a bone from a common point. Each channel has a distal end that terminates proximate the necrotic section of bone. A volume of bone growth inducing compound is introduced into each channel. The volume of bone growth inducing compound is biased toward the distal end of each channel with a screw. The screw provides immediate structural support while the bone growth inducing compound permeates the necrotic section of bone. Once the bone growth inducing compound has permeated the necrotic section of bone, it promotes rapid bone growth and thus healing of the necrotic section of bone.

Abstract: A system and method for treating a necrotic section of bone. At least one channel is drilled into a bone from a common point. Each channel has a distal end that terminates proximate the necrotic section of bone. A volume of bone growth inducing compound is introduced into each channel. The volume of bone growth inducing compound is biased toward the distal end of each channel with a screw. The screw provides immediate structural support while the bone growth inducing compound permeates the necrotic section of bone. Once the bone growth inducing compound has permeated the necrotic section of bone, it promotes rapid bone growth and thus healing of the necrotic section of bone.

Abstract: A vascular necrosis of the hip is a common disease that usually affects a young, active patient population. As the disease progresses the undermined structural integrity of the subchondral bone leads to articular collapse and subsequent osteoarthrosis. The ideal treatment is one that hinders or arrests the progression of the disease averting articular collapse and a joint replacement surgery. Several non-surgical and surgical procedures have been described to treat avascular necrosis: core decompression, osteotomies of the hip, non-vascularized and vascularized bone grafts. The purpose of this paper is to describe a surgical strategy that attempts to address the multiple factors involved in the progression of the disease: deficits in structural support, increased intraosseous pressure and the bone healing process.