Abstract: A robotic surgical system includes a master manipulator, slave robotic units having a surgical instrument for performing a Minimal Invasive Surgery (MIS), and a control system for electrically associating the master manipulator with the slave robotic units. The slave robotic unit includes the driving mechanisms which are more compact than those of the conventional MIS system. In use, the existing surgical instruments used in the conventional MIS procedure can be applied to the slave robotic unit. Moreover, by using the pivotal mechanism of the driving mechanisms, a pivot point of the surgical instrument is allowed to be shifted with respect to an incision of a patient. So, the patient's tissues surrounding the surgical instrument are not excessively affected by the surgical instrument during the procedure.

Abstract: The present application discloses monoclonal antibody against cotinine and nicotine.

Abstract: The present application discloses monoclonal antibody against cotinine and nicotine.

Abstract: A robotic surgical system includes a master manipulator, slave robotic units having a surgical instrument for performing a Minimal Invasive Surgery (MIS), and a control system for electrically associating the master manipulator with the slave robotic units. The slave robotic unit includes the driving mechanisms which are more compact than those of the conventional MIS system. In use, the existing surgical instruments used in the conventional MIS procedure can be applied to the slave robotic unit. Moreover, by using the pivotal mechanism of the driving mechanisms, a pivot point of the surgical instrument is allowed to be shifted with respect to an incision of a patient. So, the patient's tissues surrounding the surgical instrument are not excessively affected by the surgical instrument during the procedure.

Abstract: Since the K-ras oligonucleotide microarray of the present invention can detect K-ras mutations by applying a competitive DNA hybridization method to the oligonucleotides spotted on a solid matrix different from the previously reported method for detecting a mutation, it makes the more precise analysis and can reduce experimental cost and time. Accordingly, the K-ras oligonucleotide microarray of the present invention can be used in studies to detect K-ras mutations and unravel the signal transduction mechanism and tumorigenesis related to K-ras gene. Further, since the microarray of the present invention can be applied to other genes having mutational hot spot regions such as K-ras, it has wide applicable range.

Abstract: The present invention relates to a ?-catenin oligonucleotide microchip for detecting mutation in the mutational hot spot regions of ?-catenin gene, a manufacturing process thereof and a method for detecting the ?-catenin mutation employing same, wherein specific oligonucleotides are selectively designed to detect various missense mutations and in-frame deletion at the mutational hot spots of ?-catenin gene. The ?-catenin oligo chip of the present invention can be used in studies to detect ?-catenin mutations and unravel the signal transduction mechanism and tumorigenesis related to ?-catenin gene.

Abstract: The present invention relates to a ?-catenin oligonucleotide microchip for detecting mutation in the mutational hot spot regions of ?-catenin gene, a manufacturing process thereof and a method for detecting the ?-catenin mutation employing same, wherein specific oligonucleotides are selectively designed to detect various missense mutations and in-frame deletion at the mutational hot spots of ?-catenin gene. The ?-catenin oligo chip of the present invention can be used in studies to detect ?-catenin mutations and unravel the signal transduction mechanism and tumorigenesis related to ?-catenin gene.

Abstract: The present invention relates to an RET oligonucleotide microchip for detecting mutation in the mutational hot spot regions of RET gene, a manufacturing process thereof and a method for detecting hereditary cancer employing same, wherein specific oligonucleotides are selectively designed to detect various missense mutations at the mutational hot spots of RET gene.

Abstract: A human ovarian carcinoma cell line having an in-frame 3-bp deletion at codons 255 and 256 in exon 7 of the p53 gene, a nonsense mutation of codon 1815 in exon 23 of the BRCA1 gene, and a missense mutation of codon 723 in exon 19 of the hMLH1 gene.