Abstract: A circuit element comprising N paired complementary transistors. The transistors are connected to an upper (VDD) and lower voltage level (VSS), in such a way that the paired transistors operate in subthreshold. N input terminals (X1, X2 . . . XN) are connected to the respective paired transistors. Control terminals (BP, BN) are connected to control input nodes of the transistors. The circuit element provides the possibility of real time configuration between various logic functions with a minimum of transistors and wiring.

Abstract: The present invention relates to a wet acid etchant for wet acid etching of intrinsic, n-doped or p-doped Al1?x?zGaxInzAs1?ySby with 0<x<1, 0<y<1, 0?z<1 and 0<x+z<1, a process for wet acid etching of intrinsic, n-doped or p-doped Al1?x?zGaxInzAs1?ySby with 0<x<1, 0<y<1, 0?z<1 and 0<x+z<1, and a semiconductor structure prepared by wet acid etching of Al1?x?zGaxInzAs1?ySby with 0<x<1, 0<y<1, 0?z<1 and 0<x+z<1. The etchant comprises: organic acid; oxidizing agent; hydrofluoric acid.

Abstract: The present invention relates to an antibody capable of binding to human TLR2 and which (i) binds only to CD14+ cells in a normal human mononuclear cell population, and not to CD14? cells; (ii) does not inhibit LPS-induced activation of normal human mononuclear cells, and to the uses thereof, for example in the treatment of bacterial infections which are mediated via the TLR2 receptor, especially bacterial sepsis. In a particular embodiment of this aspects, a utility in the treatment of Listeria infections is proposed. The antibodies also have a utility as a general research tool, for example in screening for TLR2 expression, and in the study of TLR2 function. A further aspect of the invention provides use of an agent capable of inhibiting TLR2 activation in the preparation of a composition for the treatment of Listeria infections.