Abstract: The present disclosure embraces nucleic acid molecules found in the genome of the Citrus Leprosis Virus (CiLV), which is associated to Citrus Leprosis (CiL) disease. The cloned CiLV nucleic acid molecules can be used as probes or can be used to design oligonucleotide primers for detecting CiLV in biological samples, particularly leaves, roots and other tissues or organs of plants, such as plants from the genera Citrus and Poncirus. The cloned CiLV nucleic acid molecules are expressed in cells to provide immunogenic proteins for raising antibodies against CiLV, which can then be used to detect CiLV in biological samples. It also comprises the nucleic acid molecules represented in SEQ ID Nos. 5 and 8, in whole or part, as well as transgenic plants, such as monocots and dicots, containing the CiLV nucleic acid molecules, in any kind of combination, so that expression increases resistance to CiL disease.

Abstract: The present disclosure relates to nucleic acid molecules comprising the genome of the Citrus Sudden Death Virus (CSDV), the causative agent of Citrus Sudden Death (CSD) disease. The cloned CSDV nucleic acid molecules can be used as probes or can be used to design oligonucleotide primers useful in assays, such as a polymerase chain reaction, for detecting the presence of CSDV in biological samples, particularly leaves, roots and other tissues or organs of plants, such as plants from the genera Citrus and Poncirus. The disclosure also comprises the nucleic CSDV nucleic acid molecules, in whole or part, as well as transgenic plants, such as monocots and dicots, containing the CSDV nucleic acid molecules, in any kind of combination, so that expression increases resistance to CSD disease.

Abstract: The present invention relates to the regulation of the natural defense system of plants through the introduction of foreign/native genes into plant cells, preferably into their genomes. More specifically, the methods relate to increasing citrus plant disease resistance by over-expressing genes involved in the innate plant defense system.

Abstract: The present invention relates to methodology and constructs for modifying plant architecture and enhancing plant biomass and/or sucrose yield.

Abstract: Polynucleotides, nucleic acid constructs, and methods are disclosed for the modification of cellulose and/or lignin content in plant tissues. Plants are genetically engineered with a gene encoding a C3HC4 protein, which leads to increased cellulose content when over-expressed in the plant vascular system. Plant transformants harboring the C3HC4 protein gene show increased content of cellulose and/or decreased lignin content, traits that are thought to improve hardwood trees for cellulose extraction during pulping and papermaking.

Abstract: Polynucleotides, nucleic acid constructs, and methods are disclosed for the modification of cellulose and/or lignin content in plant tissues. Plants are genetically engineered with a gene encoding a C3HC4 protein, which leads to increased cellulose content when over-expressed in the plant vascular system. Plant transformants harboring the C3HC4 protein gene show increased content of cellulose and/or decreased lignin content, traits that are thought to improve hardwood trees for cellulose extraction during pulping and papermaking.

Abstract: The present invention relates to methodology and constructs for modifying plant architecture and enhancing plant biomass and/or sucrose yield.

Abstract: In angiosperm and gymnosperm plants, overexpressing a SAMdc nucleotide sequence can decrease lignin content and, for plants with woody tissue, increase wood density.

Abstract: The present invention relates to methodology and constructs for modifying plant architecture and enhancing plant biomass and/or sucrose yield.

Abstract: In angiosperm and gymnosperm plants, overexpressing a SAMdc nucleotide sequence can decrease lignin content and, for plants with woody tissue, increase wood density.