Abstract: Disclosed are an assembly for a surgical instrument and a surgical kit. The assembly comprises a separation member and a packaging box. The separation member and a surgical instrument switch between a first state and a second state by means of a clipping mechanism. In the first state, the separation member is connected to the surgical instrument. In the second state, the separation member is detached from the surgical instrument. The packaging box comprises an accommodation portion, and further comprises an operating member. The operating member drives the clipping mechanism to switch the separation member from the first state to the second state. The separation member is used for stripping the target tissue. The packaging box can not only provide packaging for the separation member, but also serve as a disassembling tool of the separation member relative to the surgical instrument.

Abstract: The present disclosure discloses the concept and expression method of an energy efficiency index (EEI) COPCO2 for a carbon-capture system. The COPCO2 refers to the ratio of the gain during a carbon-capture process to the cost practically, and to the ratio of the increase of CO2 chemical potential resulting from enrichment to the driving work input into the carbon-capture system physically. By introducing the concept of environmental effective state (G state), the present disclosure quantifies the “gain” during a carbon-capture process, and compares the “gain” with the “cost” to obtain the COPCO2 expression method of coefficient of performance (COP) during the carbon-capture process, which provides more accurate evaluation for the energy utilization level during a carbon-capture process and is conducive to improving the energy utilization level by carbon-capture technology, thereby improving the energy utilization rate.

Abstract: The present invention includes a method of treating a bone disease caused by a intracellular protein trafficking defect comprising: identifying a subject having the bone disease caused by the intracellular protein trafficking defect in a membrane bound transcription factor peptidase, site 1 (MBTPS1) gene; and providing the subject with an effective amount of a composition that bypasses or corrects a defect in MBTPS1 gene expression, gene splicing, or corrects protein trafficking defects in the endoplasmic reticulum and to the lysosome.