Abstract: Provided is a radiation detector with improved n/? discrimination and usable even under high counting rate conditions with a reduced load on a signal-processing system. The detector capable of distinguishing neutron and gamma-ray events includes: a scintillator; an optical filter; a first photodetector to which a first part of light emitted from the scintillator is introduced via the optical filter; and a second photodetector to which a second part of light emitted from the scintillator is introduced not via the optical filter, wherein, for a set of two wavelengths A and (A+B) nm, the scintillator emits at least a light of A nm and a light of (A+B) nm when irradiated by gamma-ray, and emits a light of A nm and does not emit a light of (A+B) nm when irradiated by neutrons; and the optical filter blocks the light of A nm and transmits the light of (A+B) nm.

Abstract: Provided is a scintillation neutron detector capable of measuring neutrons with precision even under a high amount of ? rays as background noise and excellent in neutron counting precision, the scintillation neutron detector comprising a neutron scintillator crystal containing 6Li, and the crystal having a specific surface area of no less than 60 cm2/cm3.

Abstract: The invention provides a universal linker capable of synthesizing nucleic acid having a hydroxy group at the 3? terminal, a universal support carrying the linker, and a synthesis method of nucleic acid using the universal support.

Abstract: An object of the present invention is to provide a substance which is able to be an active ingredient for the improvement of dysfunction caused by nerve damage. An improving agent for dysfunction due to nerve damage of the present invention as a means for resolution thereof is characterized in that it comprises an endo-?-N-acetylglucosaminidase type enzyme which hydrolyzes an N-acetylglucosamide bond in a keratan sulfate backbone as an active ingredient. When the improving agent of the present invention is administered, clinical improvement is achieved in motor neuron dysfunction and sensory neuron dysfunction such as neuropathic pain represented by a pain caused by allodynia and hyperalgesic reaction of the object to be treated.