Abstract: This invention relates to pharmaceutical compositions for oral administration to treat, for example, acute radiation sickness, wherein the composition comprises, for example, a first agent which is selected from the group consisting of beta-carotene, and pharmaceutically acceptable derivatives thereof; a therapeutically effective amount of a second agent which is selected from the group consisting of cholic acid, derivatives thereof, and pharmaceutically acceptable salts thereof, and at least one pharmaceutically acceptable excipient.

Abstract: The invention relates to methods for transdifferentiation of body tissues which can be used to generate specific cell types needed for regenerating organs or body parts, following cellular degeneration, injury or amputation. The present invention also describes the use of tissue transdifferentiation for treating cancer and autoimmune diseases.

Abstract: The invention relates to methods for transdifferentiation of body tissues which can be used to generate specific cell types needed for regenerating organs or body parts, following cellular degeneration, injury or amputation. The present invention also describes the use of tissue transdifferentiation for treating cancer and autoimmune disease.

Abstract: The invention relates to methods for transdifferentiation of body tissues which can be used to generate specific cell types needed for regenerating organs or body parts, following cellular degeneration, injury or amputation. The present invention also describes the use of tissue transdifferentiation for treating cancer and autoimmune disease.

Abstract: The invention relates to methods for transdifferentiation of body tissues which can be used to generate specific cell types needed for regenerating organs or body parts, following cellular degeneration, injury or amputation. The present invention also describes the use of tissue transdifferentiation for treating cancer and autoimmune diseases.

Abstract: The invention relates to methods for transdifferentiation of body tissues which can be used to generate specific cell types needed for regenerating organs or body parts, following cellular degeneration, injury or amputation. The present invention also describes the use of tissue transdifferentiation for treating cancer and autoimmune diseases.

Abstract: The invention relates to methods for transdifferentiation of body tissues which can be used to generate specific cell types needed for regenerating organs or body parts, following cellular degeneration, injury or amputation. The present invention also describes the use of tissue transdifferentiation for treating cancer and autoimmune diseases.

Abstract: The invention relates to methods for transdifferentiation of body tissues which can be used to generate specific cell types needed for regenerating a lens in the mammalian eye, following loss or removal of the original lens.

Abstract: This invention relates to methods for transdifferentiation of body tissues which can be used to generate specific cell types needed for regenerating organs or body parts, following cellular degeneration, injury or amputation. The present invention also describes the use of tissue transdifferentiation for treating cancer and autoimmune diseases.

Abstract: The invention relates to methods for transdifferentiation of body tissues which can be used to generate specific cell types needed for regenerating organs or body parts, following cellular degeneration, injury or amputation. The present invention also describes the use of tissue transdifferantiation for treating cancer and autoimmune diseases.

Abstract: Methods for raising or maintaining the level of T4 cells in humans having a depressed T4 cell level and particularly in humans infected with HIV are provided. In these embodiments, T4 enhancing amounts of beta-carotene are administered. Methods are also provided for lowering or maintaining the levels of neopterin and/or &bgr;2-microglobulin in humans having elevated levels of these indices and particularly in those infected with HIV. Beta-carotene is administered, respectively, in neopterin and &bgr;2-microglobulin therapeutically effective amounts. Also contemplated by the present invention are methods for the prevention and/or treatment of HIV infection in humans. An HIV infection preventing or an anti-HIV therapeutically effective amount of beta-carotene is administered. In another embodiment, methods are provided for preventing or treating AIDS in humans. An AIDS preventing amount or an anti-AIDS therapeutically effective amount of beta-carotene is administered.

Abstract: A method for preventing acetaminophen toxicity in a mammal is provided. An acetaminophen toxicity inhibiting amount of beta-carotene is administered before, simultaneously with, after, or any combination thereof, administering acetaminophen to the mammal. Methods for reducing acetaminophen toxicity in a mammal in need of such treatment and for increasing the maximum amount of acetaminophen that can be administered to a mammal without the mammal exhibiting acetaminophen toxicity are also provided. Acetaminophen toxicity reducing amounts and acetaminophen toxicity preventing amounts, respectively, are administered. In another embodiment, a non-steroidal analgesic and anti-inflammatory composition of acetaminophen and an acetaminophen toxicity preventing amount of beta-carotene is provided.

Abstract: According to the present invention, there are provided methods for treating age related macular degeneration in a mammal; for preventing impairment of the vision or for improving impaired vision of a mammal whose eye has drusen; for preventing formation or growth of drusen in the eye of a mammal; and for reducing the number or the size of drusen or for fading drusen without resultant areas of retinal atrophy, without resultant impairment of vision, or without a combination of the foregoing in the eye of a mammal. Beta-carotene in appropriate amounts is administered to the mammal.

Abstract: According to the present invention, there is provided a method for treating chronic lymphocytic leukemia (CLL) in a mammal. Beta-carotene is administered to the mammal in CLL therapeutically effective amounts. The invention also contemplates a method for treating T-cell chronic lymphocytic leukemia (T-CLL). T-CLL therapeutically effective amounts of beta-carotene are administered. In further embodiment, a method for lowering or maintaining the level of white blood cells in a mammal having chronic lymphocytic leukemia is provided. White blood cell therapeutically effective amounts of beta-carotene are administered to the mammal.

Abstract: According to the present invention, there are provided methods for treating age related macular degeneration in a mammal; for preventing impairment of the vision or for improving impaired vision of a mammal whose eye has drusen; for preventing formation or growth of drusen in the eye of a mammal; and for reducing the number or the size of drusen or for fading drusen without resultant areas of retinal atrophy, without resultant impairment of vision, or without a combination of the foregoing in the eye of a mammal. Beta-carotene in appropriate amounts is administered to the mammal.

Abstract: A method for preventing acetaminophen toxicity in a mammal is provided. An acetaminophen toxicity inhibiting amount of beta-carotene is administered before, simultaneously with, after, or any combination thereof, administering acetaminophen to the mammal. Methods for reducing acetaminophen toxicity in a mammal in need of such treatment and for increasing the maximum amount of acetaminophen that can be administered to a mammal without the mammal exhibiting acetaminophen toxicity are also provided. Acetaminophen toxicity reducing amounts and acetaminophen toxicity preventing amounts, respectively, are administered. In another embodiment, a non-steroidal analgesic and anti-inflammatory composition of acetaminophen and an acetaminophen toxicity preventing amount of beta-carotene is provided.