Abstract: Provided is a chemical material leakage alarm-providing method of detecting chemical material leakage in a monitoring area and providing a user alert, the method including the steps of: sensing external environmental information using an environmental information-collecting sensor; obtaining data about a chemical material using a chemical material-detecting device; predicting a diffusion pattern of the chemical material based on the environmental information; detecting chemical material information based on the chemical material data; and providing a user alert based on at least one of the diffusion pattern of the chemical material and the chemical material information.

Abstract: Provided is a chemical material leakage alarm-providing method of detecting chemical material leakage in a monitoring area and providing a user alert, the method including the steps of: sensing external environmental information using an environmental information-collecting sensor; obtaining data about a chemical material using a chemical material-detecting device; predicting a diffusion pattern of the chemical material based on the environmental information; detecting chemical material information based on the chemical material data; and providing a user alert based on at least one of the diffusion pattern of the chemical material and the chemical material information.

Abstract: The present invention relates to a use of a cell adhesion molecule-related/down-regulated by oncogene (Cdo) protein or a gene encoding the same for preventing or treating neuromuscular diseases caused by damage to motor neurons, particularly muscle stem cells and neuromuscular junctions, due to aging, oxidative stress, and the like; and a method for screening for candidates for activating the expression of Cdo. When Cdo is deleted in motor neurons or neuromuscular junctions, aggravation of damage to degenerative motor neurons due to aging, and DNA damage caused by oxidative stress and inflammation are caused, which may cause neuromuscular diseases. Thus, the diseases can be treated through overexpression and activity of the Cdo protein and the gene encoding the same.

Abstract: The present invention relates to a use for, by using the protein arginine methyltransferase 1 (PRMT1) protein or a gene encoding the same, preventing or treating a neuromuscular disease induced by motor neurons, particularly, damage to motor neurons caused by oxidative stress in the neuromuscular junction; and a method for screening a candidate material for activating the expression of PRMT1. PRMT1 deficiency in the motor nerve or neuromuscular junction induces aggravated degenerative motor nerve damage caused by aging, and DNA damage caused by oxidative stress and inflammation, thereby enabling the induction of neuromuscular disease, and thus the disease may be treated through the overexpression and activity of the PRMT1 protein and a gene encoding the same.

Abstract: A plasma enhanced vapor deposition apparatus includes a process chamber including a first space and a second space, a substrate holder provided in the first space and supporting a substrate, a plasma generating device combined to the process chamber and inducing plasma in the second space, an ion species screening member separating the first and second spaces from each other and filtering ion species to prevent the ion species from diffusing from the second space to the first space, a first gas supplier supplying a first process gas including a precursor gas into the first space, wherein the precursor gas includes atoms constituting a material layer deposited over the substrate, a second gas supplier supplying a second process gas including a reactive gas into the second space, and a gas discharger coupled to the process chamber and inducing a gas flow from the second space to the first space.