Abstract: An anticancer adjuvant for treating anticancer-agent-resistant cancer includes a tristetraprolin inhibitor. If resistance to DNA replication inhibitory anticancer agents is exhibited, a substance for inhibiting the activity of tristetraprolin can be included as an anticancer adjuvant so as to maximize the effects of an anticancer agent, and thus is usable in cancer treatment.

Abstract: A pharmaceutical composition containing a lung cancer therapeutic agent and an SIRT inhibitor may minimize the expression of the resistance of lung cancer cells against the lung cancer therapeutic agent, thereby exerting an excellent lung cancer therapeutic effect. The treatment of lung cancer cells with a lung cancer therapeutic agent and the measurement of a level of SIRT1 expression may provide information for determining whether lung cancer cells have developed chemoresistance to a lung cancer therapeutic agent, and may screen lung cancer therapeutic agents.

Abstract: Disclosed is a method for providing information used for comparing restoration rates of damaged DNA, wherein information about a time period when Ataxia telangiectasia and Rad3 related (‘ATR’) activation is accelerated, on the basis of alternative information about an expression level of cryptochrome may be acquired, therefore, it can be determined that a restoration rate of damaged DNA is high at a time period when the expression level of cryptochrome is high. Accordingly, a time period when a restoration rate of DNA damaged by different causes is high, can be determined. Further, it is possible to estimate a time period when side effects occurring due to using an anticancer drug are minimized, and then, utilize the estimated result in determining the timing of administration of an anticancer drug.

Abstract: A method of selectively killing cancer cells uses a low-temperature plasma jet device. When cancer cells are simultaneously treated with ATR and PARP-1 inhibitors, followed by synchronization of a circadian rhythm and treatment with low-temperature atmospheric-pressure plasma, cancer cell death may be maximized about ten-fold or more compared to when treated with existing low-temperature atmospheric-pressure plasma alone, and thus this method may be usefully used as a future tumor treatment method.

Abstract: Disclosed is a method for providing information used for comparing restoration rates of damaged DNA, wherein information about a time period when Ataxia telangiectasia and Rad3 related (‘ATR’) activation is accelerated, on the basis of alternative information about an expression level of cryptochrome may be acquired, therefore, it can be determined that a restoration rate of damaged DNA is high at a time period when the expression level of cryptochrome is high. Accordingly, a time period when a restoration rate of DNA damaged by different causes is high, can be determined. Further, it is possible to estimate a time period when side effects occurring due to using an anticancer drug are minimized, and then, utilize the estimated result in determining the timing of administration of an anticancer drug.

Abstract: The present disclosure relates to a novel function of a specific gene and has an effect of providing a composition for diagnosis of recurrence of bladder cancer, particularly, non-muscle invasive bladder cancer (NMIBC). Further, according to the present disclosure, it is possible to accurately predict recurrence after surgical tumor removal form a patient with non-muscle invasive bladder cancer, and the present disclosure has an effect of providing information of personalized medicine after surgical tumor removal form a patient with non-muscle invasive bladder cancer.