Abstract: Methods, systems, and computer readable media for utilizing ultrasound multiple scattering to assess pulmonary fibrosis are disclosed. One method may comprise transmitting ultrasound energy into the lungs of a subject, detecting portions of the ultrasound energy backscattered by structures in the lungs of the subject, calculating a transport parameter from the detected portions of the ultrasound energy backscattered by the structures in the lungs of the subject, inputting the transport parameter into a model that correlates values of the transport parameter with quantitative indications of severity of pulmonary fibrosis, and receiving, from the model, a quantitative indication of the severity of pulmonary fibrosis for the subject that correlates with the transport parameter for the subject.

Abstract: Methods, systems, and computer readable media for utilizing ultrasound multiple scattering to assess pulmonary fibrosis are disclosed. One method may comprise transmitting ultrasound energy into the lungs of a subject, detecting portions of the ultrasound energy backscattered by structures in the lungs of the subject, calculating a transport parameter from the detected portions of the ultrasound energy backscattered by the structures in the lungs of the subject, inputting the transport parameter into a model that correlates values of the transport parameter with quantitative indications of severity of pulmonary fibrosis, and receiving, from the model, a quantitative indication of the severity of pulmonary fibrosis for the subject that correlates with the transport parameter for the subject.

Abstract: Methods are described for reducing cytoskeletal rearrangement and intercellular gap formation by contacting cells with an aminoalkyl glucosaminide phosphate. In particular, the methods can be used to reduce actin cytoskeletal rearrangement and/or intracellular gap formation related to ischemic or ischemia-reperfusion events and to alleviate diseases or conditions related to increased actin cytoskeletal rearrangement.

Abstract: Methods are described for preventing or reducing edema related to ischemia-reperfusion by treating the organ or tissue being transplanted with an aminoalkyl glucosaminide phosphate.

Abstract: Methods are described for preventing or reducing cytoskeletal rearrangement and intercellular gap formation by contacting cells with an aminoalkyl glucosaminide phosphate. In particular, the disclosed methods can be used to prevent or reduce actin cytoskeletal rearrangement and/or intracellular gap formation related to ischemic or ischemia-reperfusion events and to prevent or alleviate diseases or conditions related to increased actin cytoskeletal rearrangement.