Abstract: A payment interface display method and apparatus and an electronic device are provided. The payment interface display method includes: receiving a first input; displaying a first interface in response to the first input, where the first interface includes at least one payment control, and each payment control is associated with a payment function interface; receiving a second input for a target payment control, where the target payment control is a payment control of the at least one payment control; and displaying, in response to the second input, the target payment function interface associated with the target payment control.

Abstract: A tensegrity offshore wind power generation support structure is provided, relating to the technical field of offshore wind power. The support structure includes inclined columns, prestressed cables, a rigid support, a floating foundation and anchoring systems. A stable self-balancing space supporting structure is formed by the inclined columns and the cables; the inclined columns inclines outwards, upper parts of the inclined columns are connected with the prestressed cables; the bottom ends of the inclined columns are connected with the floating foundation; the middle parts of the inclined columns are connected with the rigid support; and the floating foundation is fixed with a seabed through the anchoring systems. According to the support structure, a tower in the traditional design is not needed, and all the cables are ensured to be in a tension state through the support of the inclined columns.

Abstract: A payment interface display method and apparatus and an electronic device are provided. The payment interface display method includes: receiving a first input; displaying a first interface in response to the first input, where the first interface includes at least one payment control, and each payment control is associated with a payment function interface; receiving a second input for a target payment control, where the target payment control is a payment control of the at least one payment control; and displaying, in response to the second input, the target payment function interface associated with the target payment control.

Abstract: A method for regulating Src and its downstream signaling pathway which includes binding between Src and Na+/K+-ATPase is disclosed. The Na+/K+-ATPase/Src complex is a functional receptor for cardiotonic steroids such as ouabain. Also disclosed are Src inhibitors or activators which include either Na+/K+-ATPase or Src that interfere with the interaction between the Na/K-ATPase and Src, act via a different mechanism from ATP analogues, and is pathway (Na+/K+-ATPase) specific. Also disclosed is use of said Src-modulating peptides for treatment of cancer.

Abstract: A novel Src inhibitor that targets the Na/K-ATPase/Src receptor complex and antagonizes ouabain-induced protein kinase cascades and uses thereof are disclosed.

Abstract: A method for regulating Src and its downstream signaling pathway which includes binding between Src and Na+/K+-ATPase is disclosed. The Na+/K+-ATPase/Src complex is a functional receptor for cardiotonic steroids such as ouabain. Also disclosed are Src inhibitors or activators which include either Na+/K+-ATPase or Src that interfere with the interaction between the Na/K-ATPase and Src, act via a different mechanism from ATP analogues, and is pathway (Na+/K+-ATPase) specific.

Abstract: A method for regulating Src and its downstream signaling pathway which includes binding between Src and Na+/K+-ATPase is disclosed. The Na+/K+-ATPase/Src complex is a functional receptor for cardiotonic steroids such as ouabain. Also disclosed are Src inhibitors or activators which include either Na+/K+-ATPase or Src that interfere with the interaction between the Na/K-ATPase and Src, act via a different mechanism from ATP analogs, and is pathway (Na+/K+-ATPase) specific.

Abstract: A novel Src inhibitor that targets the Na/K-ATPase/Src receptor complex and antagonizes ouabain-induced protein kinase cascades and uses thereof are disclosed.

Abstract: Described herein are methods to bind a compound to the SH2 domain of Src in a Src-expressing cell which includes contacting a compound comprising an amino acid compound to a Src-expressing cell; and binding a compound to the SH2 domain of Src.

Abstract: This invention is based in part on the elucidation of new structural conformations and functions of the sodium/potassium adenosine triphosphate synthase (Na/K ATPase), and especially elucidation of new binding sites and interactions. The present invention provides practical applications of several surprising structural and functional relationships between Na/K ATPase and compounds which interact with Na/K ATPase. Disclosure of these structures and relationships provides insight and practical solutions to chemically affecting not only the Na/K ATPase interactions, but also regulators known to be upstream and downstream.

Abstract: This invention is based in part on the elucidation of new structural conformations and functions of the sodium/potassium adenosine triphosphate synthase (Na/K ATPase), and especially elucidation of new binding sites and interactions. The present invention provides practical applications of several surprising structural and functional relationships between Na/K ATPase and compounds which interact with Na/K ATPase. Disclosure of these structures and relationships provides insight and practical solutions to chemically affecting not only the Na/K ATPase interactions, but also regulators known to be upstream and downstream.

Abstract: A novel Src inhibitor that targets the Na/K-ATPase/Src receptor complex and antagonizes ouabain-induced protein kinase cascades and uses thereof are disclosed.