Abstract: An IC package is assembled from a bumpless die, a die carrier having a plurality of solder bumps thereon, and a heat spreader lid. The bumpless die is bonded to the heat spreader lid to form a module and then the module is bonded to the bumped die carrier.

Abstract: An IC package is assembled from a bumpless die, a die carrier having a plurality of solder bumps thereon, and a heat spreader lid. The bumpless die is bonded to the heat spreader lid to form a module and then the module is bonded to the bumped die carrier.

Abstract: Disclosed is a null mutant (or knockout) rodent comprising in its germ cells an artificially induced PTTG null mutation. In some embodiments, the null mutant rodent can be generated by way of homologous recombination in an embryonic stem cell or germ cell. The inventive null mutant rodent can be used to study mammalian physiology at the cellular, tissue, and/or organismal level with respect to various phenotypes, including hyperglycemia, hypoinsulinaemia, hypoleptinemia, diabetes, chromosomal aneuploidy, premature centromere division, chromosomal damage, aberrant mitotic cellular division, thrombocytopenia, thymic hyperplasia, splenic hypoplasia, testicular hypoplasia, and female subfertility. Also disclosed is an animal model for diabetes. Also disclosed is a somatic or germ cell obtained from the null mutant rodent. Also disclosed is a cell line derived from a cell obtained from the null mutant rodent.

Abstract: The present invention provides a BZR1 gene and an altered gene, bzr1-D, involved in the brassinosteroid response pathway. The expression of BZR1 genes in transgenic plants causes such plants to become significantly larger and more robust than their wild-type counterparts, thus increasing plant yields.

Abstract: Disclosed is a null mutant (or knockout) rodent comprising in its germ cells an artificially induced PTTG null mutation. In some embodiments, the null mutant rodent can be generated by way of homologous recombination in an embryonic stem cell or germ cell. The inventive null mutant rodent can be used to study mammalian physiology at the cellular, tissue, and/or organismal level with respect to various phenotypes, including hyperglycemia, hypoinsulinaemia, hypoleptinemia, diabetes, chromosomal aneuploidy, premature centromere division, chromosomal damage, aberrant mitotic cellular division, thrombocytopenia, thymic hyperplasia, splenic hypoplasia, testicular hypoplasia, and female subfertility. Also disclosed is an animal model for diabetes. Also disclosed is a somatic or germ cell obtained from the null mutant rodent. Also disclosed is a cell line derived from a cell obtained from the null mutant rodent.

Abstract: The present invention provides systems and methods for large-scale genetic measurements by generating from a sample labeled target sequences whose length, orientation, label, and degree of overlap and complementarity are tailored to corresponding end-attached probes of a solid support so that signal-to-noise ratios of measurement from specifically hybridized labeled target sequences are maximized. Systems for implementing methods of the invention include a set of sample-interacting probes to produce amplicons that either each contain a segment of a target polynucleotide or an oligonucleotide tag that corresponds to a segment of a target polynucleotide, one or more solid phase supports that contain a plurality of end-attached probes, and methods of generating from sample-interacting probe amplicons from which labeled target sequences are tailored for hybridization to the solid phase supports, such as microarrays.

Abstract: The invention provides an electronic assembly comprising a carrier substrate, a die, and a solidified underfill material. The carrier substrate has an upper plane. The die has a die substrate and an integrated circuit formed on one side of the die substrate. The die has a lower major surface over the upper plane, an upper major surface, and a plurality of side edge surfaces from the upper major surface to the lower major surface. A corner edge portion where extensions of two of the side edge surfaces meet has been removed. The solidified underfill material is located between and contacts both the upper plane of the carrier substrate and the lower surface of the die.

Abstract: A latch needle (1) has a latch spring (19), which assumes the width of the latch slit (5) in the vicinity of the latch seating (18). However, the latch spring (19) is embodied to be narrower on both sides of this section (25). By means of this, premature wear of the latch or spring in the area of the latch seating device (18) is counteracted on the one hand as is, on the other, the obstruction of the latch slit (5) by dust or dirt. The narrower sections (26, 27) of the latch spring (19) define a passage together with the inner face (8, 9) of the slit walls (6, 7), through which foreign bodies, such as fiber remnants, dust or the like can be conveyed from the upper needle side (11) in the direction toward the needle back (12). In contrast to conventional latch needles, needles (1) of this type of construction show reduced wear, a reduced susceptibility to becoming soiled, and therefore increased dependability.

Abstract: The present invention provides a cytochrome P450-encoding gene, DAS5, as well as an altered gene, das5-D, which are involved in the synthesis of the plant steroid hormone brassinolide. The das5-D mutant has an insertion of a T-DNA containing transcriptional enhancers into its promoter region. Overexpression of the mutant thus produces large amounts of DAS5 transcript. The expression of this das5-D mutation or overexpression of the DAS5 gene in transgenic plants causes such plants to become significantly larger and more robust than their wild-type counterparts, thus increasing plant yields.

Abstract: A latch needle (1) has a latch spring (19), which assumes the width of the latch slit (5) in the vicinity of the latch seating (18). However, the latch spring (19) is embodied to be narrower on both sides of this section (25). By means of this, premature wear of the latch or spring in the area of the latch seating device (18) is counteracted on the one hand as is, on the other, the obstruction of the latch slit (5) by dust or dirt. The narrower sections (26, 27) of the latch spring (19) define a passage together with the inner face (8, 9) of the slit walls (6, 7), through which foreign bodies, such as fiber remnants, dust or the like can be conveyed from the upper needle side (11) in the direction toward the needle back (12). In contrast to conventional latch needles, needles (1) of this type of construction show reduced wear, a reduced susceptibility to becoming soiled, and therefore increased dependability.

Abstract: The present invention provides a BZR1 gene and an altered gene, bzr1-D, involved in the brassinosteroid response pathway. The expression of BZR1 genes in transgenic plants causes such plants to become significantly larger and more robust than their wild-type counterparts, thus increasing plant yields.

Abstract: The present invention provides a cytochrome P450-encoding gene, DAS5, as well as an altered gene, das5-D, which are involved in the synthesis of the plant steroid hormone brassinolide. The das5-D mutant has an insertion of a T-DNA containing transcriptional enhancers into its promoter region. Overexpression of the mutant thus produces large amounts of DAS5 transcript. The expression of this das5-D mutation or overexpression of the DAS5 gene in transgenic plants causes such plants to become significantly larger and more robust than their wild-type counterparts, thus increasing plant yields.

Abstract: Disclosed is a null mutant (or knockout) rodent comprising in its germ cells an artificially induced PTTG null mutation. In some embodiments, the null mutant rodent can be generated by way of homologous recombination in an embryonic stem cell or germ cell. The inventive null mutant rodent can be used to study mammalian physiology at the cellular, tissue, and/or organismal level with respect to various phenotypes, including hyperglycemia, hypoinsulinaemia, hypoleptinemia, diabetes, chromosomal aneuploidy, premature centromere division, chromosomal damage, aberrant mitotic cellular division, thrombocytopenia, thymic hyperplasia, splenic hypoplasia, testicular hypoplasia, and female subfertility. Also disclosed is an animal model for diabetes. Also disclosed is a somatic or germ cell obtained from the null mutant rodent. Also disclosed is a cell line derived from a cell obtained from the null mutant rodent.

Abstract: A method and apparatus for processing and encoding video data is presented that allocates available bandwidth, and hence image quality, in dependence upon the relative speed of motion of objects in a sequence of images forming the video data. Fast moving objects are allocated less quality, or precision, than slower moving or stationary objects. In a preferred embodiment of this invention, the quantization step size is dependent upon the magnitude of the motion vector associated with each block in each frame of a video sequence. In a further embodiment of this invention, the quantization step size is also dependent upon the location of each block in each frame, providing more precision to a central area of each frame. To reduce computational complexity, a motion activity map is created to identify areas of higher precision based upon the location and motion associated with each block.

Abstract: Disclosed is a cap-like reservoir defining system which allows user adjustable location of application of cutting tool lifetime increasing cryogenic cooling to a workpiece contacting edge of a cutting tool, in a workpiece machining system. The present invention method of use finds application in the machining of materials such as ceramics, heat resistant materials, titanium, inconel alloys, and super alloys.